Katherine Weilbaecher, MD

Katherine Weilbaecher

Researcher Background

Primary Academic Title

Professor of Medicine, Division of Oncology, Section of Molecular Oncology, Washington University School of Medicine

Training

  • 1992 - 1994: Intern and resident, internal medicine, Stanford University Hospital, Stanford, CA
  • 1994 - 1998: Fellow, medical oncology, Dana-Farber Cancer Institute, Boston, MA
  • 1994 - 1998: Clinical fellow, internal medicine, Brigham and Women's Hospital and Harvard Medical School, Boston, MA

Education

  • 1992: MD, Stanford University, Stanford, Calif.

Research & Selected Publications

Research Interest

Osteoclasts are central to the pathogenesis of skeletal metastases. Pharmacologic blockade of osteoclast function and development using bisphosphonates can result in decreases in bone metastases, fractures and bone pain. My laboratory studies the transcriptional regulation of osteoclast development to identify novel targets for therapeutic intervention in bone metastases from breast and prostate cancer. The Microphthalmia transcription factor (Mitf) and the related protein TFE3 are critical to osteoclast fusion and maturation. We study the signaling of Macrophage colony stimulating factor (M-CSF) and RANKL, two vital osteoclastogenic factors, with the Microphthalmia transcription factor family and its nuclear target genes. We are also developing animal models of bone metastases using osteopetrotic mouse mutants and other genetically targeted osteoclast mutants. Key words: Breast cancer, bone metastases, osteoclast development, beta 3 integrin signaling, microphthalmia transcription factor, RANKL signaling, osteolytic and osteoblastic bone metastases, bone marrow metastases.

Detection of Circulating Tumor DNA Using a Tissue-Free Epigenomic Assay Is a Highly Prognostic Biomarker in Early-Stage Triple-Negative Breast Cancer.
Authors:

Ademuyiwa FO, Ma CX, Weilbaecher K, one or more additional authors omitted Davis A

Journal & Year:

Clin Cancer Res • 2025

HTLV-1 infected T cells cause bone loss via small extracellular vesicles.
Authors:

Pokhrel NK, Panfil AR, Habib H, one or more additional authors omitted Weilbaecher KN, Veis DJ

Journal & Year:

J Extracell Vesicles • 2024

Chemotherapy and the Extra-Tumor Immune Microenvironment: EXTRA-TIME.
Authors:

Phan TG, Weilbaecher KN, Aft R, Croucher PI , Chaffer CL

Journal & Year:

Cancer Discov • 2024